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The clinical outcomes of nocturnal hypoxaemia in patients with COPD and daytime PaO2 ≥sixty mmHg (8.0 kPa) are unclear. Even though it's been proposed that repetitive transient desaturations throughout sleep could be one particular system underlying the development of pulmonary hypertension in COPD, the primacy of hypoxia being a driving force in
The clinical consequences of nocturnal hypoxaemia in patients with COPD and daytime PaO2 ≥sixty mmHg (8.0 kPa) are unclear. While it has been instructed that repetitive transient desaturations through sleep could possibly be one particular system underlying the development of pulmonary hypertension in COPD, the primacy of hypoxia for a driving fo